

S454
24th European Congress of Psychiatry / European Psychiatry 33S (2016) S349–S805
before obtaining diagnosis of FTD. There is no conclusive evidence
about the anatomical correlation of psychotic features in the FTD,
although there is the possible association with the right brain
degeneration.
Conclusions
The existence of psychotic symptoms do not argues
against the diagnosis of FTD verifying a high frequency of psychosis
in FTD – C9
+
. As can be the first symptom in FTD is critical to differ-
entiate psychiatric disorders. Further studies are needed in order
to obtain a better characterization of psychotic symptoms in FTD –
C9
+
.
Disclosure of interest
The authors have not supplied their decla-
ration of competing interest.
http://dx.doi.org/10.1016/j.eurpsy.2016.01.1315EV331
Relationship between emotional
intelligence and neurocognition in
severe mental disorders
E. Chapela
1 ,∗
, I. Morales
2, J. Quintero
1 , 2, M. Félix-Alcántara
1,
J. Correas
3, J. Gómez-Arnau
31
Hospital Universitario Infanta Leonor, Psychiatry, Madrid, Spain
2
Psikids, Psychiatry, Madrid, Spain
3
Hospital del Henares, Psychiatry, Madrid, Spain
∗
Corresponding author.
Introduction
The severemental disorders are the subject of grow-
ing research in the area of emotional intelligence because of his
relationship with psychosocial functionality loss. Despite treat-
ment advances, patients continue to experience high levels of
social, professional and personal disabilities, related to the pres-
ence of deficits in cognition. These changes are manifested in two
areas: the neurocognitive and social cognition.
Objectives
To better understand the relationship between neuro-
and sociocognition in schizophrenia and bipolar disorder.
Aims
The aim of this research is to study the factors related to
emotional intelligence, with particular interest in neurocognitive
deficits.
Methods
A total of 75 adult patients with schizophrenia and
bipolar disorder type I were evaluated. The assessment protocol
consisted of a questionnaire on socio-demographic and clinical-
care data, and a battery of clinical and cognitive scales, including
MSCEIT, WAIS-IV, TMT and Rey Figure.
Results
MSCEIT was negatively correlated with age, the severity
of the clinical symptoms (BPRS, CGI-S), the TMT-A and the Test of
Complex Figure, and positively with the intelligence quotient.
Conclusions
The deficits in emotional intelligence are part of a set
of cognitive, social and non-social skills, which are altered in these
severe mental disorders. Emotional intelligence worsens with the
deterioration of cognitive functioning, executive dysfunction and
severity of psychiatric disorder.
Disclosure of interest
The authors have not supplied their decla-
ration of competing interest.
http://dx.doi.org/10.1016/j.eurpsy.2016.01.1316EV332
Are we able to influence cognitive
dysfunction in multiple sclerosis?
E.I. Davidescu
1 ,∗
, S .A. Nicolae
1 , I. Buraga
1 , C. Tudose
2 , N.Popa
31
University of Medicine and Pharmacy “Carol Davila”, Colentina
Clinical Hospital, Neurology, Bucharest, Romania
2
University of Medicine and Pharmacy “Carol Davila”, Alexandru
Obregia Clinical Hospital of Psychiatry, Psychiatry, Bucharest,
Romania
3
Alexandru Obregia Clinical Hospital of Psychiatry, Psychiatry,
Bucharest, Romania
∗
Corresponding author.
Introduction
Multiple sclerosis (MS) is the most common chronic
neurologic disease affecting young people. Cognitive dysfunction is
an important part of disability, interfering with quality of life (QoL).
Disease modifying therapies (DMT) are gold standard of long-term
treatment in MS.
Objectives
Assessment of DMT impact on evolution of cognitive
dysfunction.
Aims
To analyze the cognitive status in a lot of 74 patients with
MS, with a mean age of 40.4 years, treated with different DMT in
the National Health Program.
Methods
Testing patients during 2014–2015 for cognitive dys-
function, by applying MMSE, Sunderland Clock Test, Beck
Depression Inventory, Fatigue Impact Scale and QoL Short form-36
scores every 6months; analyzing demographic, clinical and mag-
netic resonance imagery (MRI) data.
Results
Thirty-six percent of lot showed memory and concen-
tration changes (12 patients with secondary progressive MS, 15
with relapsing-remitting MS); mean age of these patients was
46.29 years, with a mean period of evolution of the disease of
9.8 years before starting DMT; cortical atrophy was present on MRI
in 37% of these patients. Mean age of those who didn’t present
cognitive disturbances was 37.01 years, with a mean period of
evolution of 6.2 years before starting DMT. Disturbances appeared
independently of the presence of cortical atrophy, as this marker
appeared in 5% of patients with no cognitive dysfunction.
Conclusions
When starting DMT, age and time of evolution of the
disease are essential for further developing of cognitive dysfunc-
tion. Mood and anxiety disturbances can be a prodromal marker
of neurocognitive troubles. DMT have neuroprotective outcome in
MS.
Disclosure of interest
The authors have not supplied their decla-
ration of competing interest.
http://dx.doi.org/10.1016/j.eurpsy.2016.01.1317EV333
The effect of interpersonal
multisensory stimulation on the
self-face recognition in adults with
autistic syndrome disorder
N. Deltort
1 , 2 ,∗
, J.R. Cazalets
2, A. Amestoy
1 , 2, M. Bouvard
1 , 21
Centre hospitalier Charles-Perrens, centre ressource autisme,
Bordeaux, France
2
CNRS, INCIA, UMR 5287, Bordeaux, France
∗
Corresponding author.
Studies on individuals without developmental disorder show that
mental representation of self-face is subject to a multimodal pro-
cess in the same way that the representation of the self-body is.
People with autistic spectrum disorder (ASD) have a particular pat-
tern of face processing and a multimodal integration deficit.
The objectives of our study were to evaluate the self-face recogni-
tion and the effect of interpersonal multisensory stimulation (IMS)
in individuals with ASD. We aimed to show a self-face recognition
deficit and a lack of multimodal integration among this population.
IMS consisted of the presentation of a movie displaying an unfamil-
iar face being touched intermittently, while the examiner applied
the same stimulation synchronously or asynchronously on the par-
ticipant. The effect resulting from IMSwasmeasured on two groups
with or without ASD by a self-face recognition task on morphing
movies made from self-face and unfamiliar-face pictures.
There was a significant difference between groups on self-
recognition before stimulation. This result shows a self-face
recognition deficit in individuals with ASD. Results for the control
group showed a significant effect of IMS on self-face recognition in
synchronous condition. This suggests the existence of an update of
self-face mental representation bymultimodal process. In contrast,
there was no significant effect of IMS demonstrated in ASD group,